CIFOR-ICRAF berfokus pada tantangan-tantangan dan peluang lokal dalam memberikan solusi global untuk hutan, bentang alam, masyarakat, dan Bumi kita

Kami menyediakan bukti-bukti serta solusi untuk mentransformasikan bagaimana lahan dimanfaatkan dan makanan diproduksi: melindungi dan memperbaiki ekosistem, merespons iklim global, malnutrisi, keanekaragaman hayati dan krisis disertifikasi. Ringkasnya, kami berupaya untuk mendukung kehidupan yang lebih baik.

CIFOR-ICRAF menerbitkan lebih dari 750 publikasi setiap tahunnya mengenai agroforestri, hutan dan perubahan iklim, restorasi bentang alam, pemenuhan hak-hak, kebijakan hutan dan masih banyak lagi – juga tersedia dalam berbagai bahasa..

CIFOR-ICRAF berfokus pada tantangan-tantangan dan peluang lokal dalam memberikan solusi global untuk hutan, bentang alam, masyarakat, dan Bumi kita

Kami menyediakan bukti-bukti serta solusi untuk mentransformasikan bagaimana lahan dimanfaatkan dan makanan diproduksi: melindungi dan memperbaiki ekosistem, merespons iklim global, malnutrisi, keanekaragaman hayati dan krisis disertifikasi. Ringkasnya, kami berupaya untuk mendukung kehidupan yang lebih baik.

CIFOR–ICRAF publishes over 750 publications every year on agroforestry, forests and climate change, landscape restoration, rights, forest policy and much more – in multiple languages.

CIFOR–ICRAF addresses local challenges and opportunities while providing solutions to global problems for forests, landscapes, people and the planet.

We deliver actionable evidence and solutions to transform how land is used and how food is produced: conserving and restoring ecosystems, responding to the global climate, malnutrition, biodiversity and desertification crises. In short, improving people’s lives.

bta-microRNA-145 1 - 3 - B / (In Chinese )

Ekspor kutipan

The object of this experiment was to study the effects of bta-microRNA-145 (bta-miR-145) on the expressions of genes related to insulin-like growth factor 1 receptor-phosphatidylinositol 3-kinase-protein kinase B-mammalian target of rapamycin (IGF1R-PI3K-Akt/mTOR) signaling pathway, and to provide the basis for the regulation of lactation in dairy cow from the perspective of the microRNA. Primary bovine mammary epithelial cells were selected as research object. bta-miR-145 mimic and inhibitor were transfected into cells. After transfection, the expression changes of 15 genes related to IGF1R-PI3K-Akt/mTOR signaling pathway were detected by real-time qPCR. The results showed that the over-expression or down-expression of bta-miR-145 did not cause the expression change of predicted target IGF1R, insulin receptor substrate 1 (IRS1) and beta-casein (CSN2) (P>0.05); the over-expression of bta-miR-145 significantly caused down-regulation of eukaryotic translation initiation factor 4E (EIF4E) mRNA expression (P

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